Plant Comparison

Perforate St John’s-wort vs Chickweed

A side-by-side comparison of two medicinal plants — every documented constituent, action, use, safety note and cited source, assembled automatically from the Omnia Sana database.

First plant
Second plant
Show:
Plant APerforate St John’s-wortHypericum perforatumHypericaceaeFull monograph →
Plant BChickweedStellaria mediaCaryophyllaceaeFull monograph →

At a glance

Perforate St John’s-wort and Chickweed: they share 4 indicated uses (arthritis / joint pain, eczema, inflammation (general), …); 3 pharmacological actions in common.

Perforate St John’s-wortChickweed
Constituents33
Pharmacological actions63
Indicated uses94
Safety notes22
Cited sources3120
Indicated uses
Only Perforate St John’s-wort
BruisingCold & fluInfection (general)Insomnia / sleeplessnessWounds
Shared (4)
Arthritis / joint painEczemaInflammation (general)Skin irritation
Only Chickweed
none
Pharmacological actions
Only Perforate St John’s-wort
AntiviralSedative / sleep supportVulnerary (wound healing)
Shared (3)
Anti-inflammatoryAntioxidantEmollient / skin-soothing
Only Chickweed
none

Evidence face-off — shared uses

ConditionPerforate St John’s-wortChickweedVerdict
Arthritis / joint pain1/102/10Comparable evidence
Eczema1/101/10Comparable evidence
Inflammation (general)1/102/10Comparable evidence
Skin irritation1/101/10Comparable evidence

Evidence scores (1–10) are computed from the tier of each cited source. “Comparable” means the two scores are within one point. Follow a score to its detailed sources.

Key Constituents

Naphthodianthrones (hypericin, pseudohypericin)[4]

Red pigments historically used to standardise extracts; contribute to antiviral activity and are the compounds responsible for photosensitivity risk.

Phloroglucinols (hyperforin)[4]

Considered the principal compound behind the antidepressant activity and the main driver of the herb's potent CYP3A4/P-glycoprotein induction and drug-interaction profile.

Hyperforin
Flavonoids (hyperoside, quercetin, rutin)[4]

Antioxidant flavonoids contributing to overall activity.

FlavonoidsQuercetinRutin
Flavonoids and coumarins[1]

Contribute to antioxidant and anti-inflammatory activity.

FlavonoidsCoumarins
Saponins[1]

Minor constituents; large amounts of fresh herb have anecdotally been linked to mild gastrointestinal upset, possibly related to saponin content.

Saponins
Vitamin C and minerals[1]

Nutritive constituents supporting its traditional use as a wild edible green.

Pharmacological Actions

Anti-inflammatory[5, 15, 16]
Antioxidant[6, 15, 16]
Antiviral[15, 16]
Emollient / skin-soothing[15, 16]
Sedative / sleep support[1, 2, 4, 5, 9, 11, 12, 13, 14, 15, 16]
Vulnerary (wound healing)[4, 15, 16]
Anti-inflammatory[1, 4, 5, 7, 11, 12, 13]
Antioxidant[1, 5, 6, 11, 12, 13]
Emollient / skin-soothing[11, 12, 13]

Traditional & Indicated Uses

Arthritis / joint pain[15, 16]Traditional · 1/10

inferred from anti-inflammatory action

Evidence: 1
Label: Arthritis / joint pain
Bruising[15, 16]Traditional · 1/10

inferred from vulnerary action

Evidence: 1
Label: Bruising
Cold & flu[15, 16]Traditional · 1/10

inferred from antiviral action

Evidence: 1
Label: Cold & flu
Eczema[15, 16]Traditional · 1/10

inferred from emollient action

Evidence: 1
Label: Eczema
Infection (general)[15, 16]Traditional · 1/10

inferred from antiviral action

Evidence: 1
Label: Infection (general)
Inflammation (general)[15, 16]Traditional · 1/10

inferred from anti-inflammatory action

Evidence: 1
Label: Inflammation (general)
Insomnia / sleeplessness[15, 16]Traditional · 1/10

inferred from sedative action

Evidence: 1
Label: Insomnia / sleeplessness
Skin irritation[15, 16]Traditional · 1/10

inferred from anti-inflammatory action

Evidence: 1
Label: Skin irritation
Wounds[15, 16]Traditional · 1/10

inferred from vulnerary action

Evidence: 1
Label: Wounds
Arthritis / joint pain[7, 11, 12, 13]Traditional · 2/10

inferred from anti-inflammatory action

Evidence: 2
Label: Arthritis / joint pain
Eczema[11, 12, 13]Traditional · 1/10

inferred from emollient action

Evidence: 1
Label: Eczema
Inflammation (general)[7, 11, 12, 13]Traditional · 2/10

inferred from anti-inflammatory action

Evidence: 2
Label: Inflammation (general)
Skin irritation[11, 12, 13]Traditional · 1/10

inferred from anti-inflammatory action

Evidence: 1
Label: Skin irritation

Safety, Cautions & Contraindications

Safety note[15, 16]Caution

Significant drug interactions: strongly induces CYP3A4 and P-glycoprotein, reducing plasma levels of many drugs including oral contraceptives, antiretrovirals, cyclosporine, warfarin, and digoxin. May cause photosensitivity (fair-skinned individuals). Do not combine with SSRIs, SNRIs, or MAOIs (serotonin syndrome risk). Avoid in bipolar disorder.

Safety note[15, 16, 17]Caution

Duke (2002) rates St. John's wort as ++ (raised from earlier editions) and notes antidepressant, antiviral, antibacterial, and wound-healing activities at experimental and clinical levels. Hypericin and hyperforin are identified as key active constituents. Clinical evidence (score 2) confirms antidepressant efficacy in mild-to-moderate depression, and WHO/Commission E provide approval for this indication. Dose: 300 mg standardized extract (0.3% hypericin, 3–5% hyperforin) three times daily. Duke emphasizes the critical drug interaction: St. John's wort is a potent CYP3A4 inducer and can significantly reduce blood levels of many medications including oral contraceptives, cyclosporine, antiretrovirals, and warfarin. Avoid combined use with SSRIs (serotonin syndrome risk) and pharmaceutical antidepressants (Duke, 2002).

Safety note[11, 12, 13]Caution

Don’t overdo it as food: some wild-edible-plant research flags chickweed among plants checked for oxalic acid / nitrates—fine in normal amounts, but “huge daily servings” aren’t a smart idea for everyone. (Guil et al., 1997). Kidney stones / high-oxalate diets: if you’re prone to calcium-oxalate stones, treat chickweed like other higher-oxalate greens—moderation. (Guil et al., 1997). Possible stomach upset in large amounts (nausea/diarrhea reported anecdotally; some sources link this to saponins and “too much fresh herb”). (Singh et al., 2022). Pregnancy / breastfeeding: because good human data is limited, many herbal references advise sticking to food amounts or avoiding medicinal dosing unless guided by a professional. (Singh et al., 2022).

Safety note[11, 12, 13, 14]Info

Duke (2002) notes that chickweed has primarily folkloric (score f) activity as a demulcent, emollient, and depurative. Its main medicinal applications are external — including soothing inflamed skin, eczema, and itching. No strong clinical evidence supports specific internal medicinal uses. The plant contains flavonoids and coumarins. It is considered generally safe as a dietary green and in topical preparations, though no standardized dosages are established (Duke, 2002).

External Ids

Gbif: 3189486
Powo: urn:lsid:ipni.org:names:433719-1
Wikidata: Q158289
Gbif: 5384604
Wikidata: Q158711

Botanical Description

Erect, branching perennial herb with paired, oval leaves dotted with tiny translucent oil glands that look like pinpricks when held to the light (giving the species name 'perforatum'). Bright yellow, five-petalled flowers with numerous stamens and black dots along the petal edges are borne in flat-topped clusters, and release a reddish pigment when crushed.[4]

Height: 30-90 cm
Habit: Erect, branching perennial herb
Leaves: Paired, oval, dotted with tiny translucent oil glands
Flowers: Bright yellow, five-petalled, with numerous stamens and black-dotted petal edges, in flat-topped clusters
Stem: Erect, branching, with two raised longitudinal ridges
Root: Woody rootstock with spreading rhizomes
Fruit: Small, three-valved capsule
Flowering Period: June-September (traditionally around St John's Day, 24 June)

Low, sprawling annual herb (Caryophyllaceae) forming mats, with weak trailing stems bearing a single line of fine hairs running up one side, switching sides at each leaf node - a key identification feature. Leaves are opposite, ovate and pointed. Tiny white, star-shaped flowers have five deeply notched petals that appear as ten, followed by a small capsule.[1]

Height: Low, sprawling/mat-forming, stems to about 40 cm
Habit: Low, sprawling, mat-forming annual herb
Leaves: Opposite, ovate, pointed
Flowers: Tiny, white, star-shaped, five deeply notched petals (appearing as ten)
Stem: Weak, trailing, with a single line of fine hairs switching sides at each node
Root: Shallow fibrous roots
Fruit: Small capsule
Flowering Period: Can flower almost year-round in mild climates, peaking spring-autumn

Habitat

Grows in grassland, roadsides, woodland clearings and waste ground on well-drained soils; native to Europe, Western Asia and North Africa and widely naturalised elsewhere, including North America and Australia.[4]

Native to Europe, now a cosmopolitan weed found worldwide, growing in disturbed ground, gardens, cultivated land and waste places, tolerating cool, moist conditions.[1]

Harvesting

The flowering tops (flower and upper leaf) are cut in summer as the flowers open, around the traditional St John's Day period, and dried quickly in a warm, shaded, airy place to preserve the hypericin and hyperforin content.[4]

Parts: Flower, Leaf
Season: Summer, at flowering

The whole aerial plant is gathered fresh, ideally in spring or autumn when most tender, and used fresh or dried.[1]

Parts: Flower, Leaf, Stem
Season: Spring or autumn, when most tender

Traditional Uses

St John's wort has a centuries-long European folk reputation as a wound-healing and nerve tonic herb, traditionally used for mild wounds, burns and nerve pain and to lift low mood; this traditional mood-supporting use is now one of the most extensively clinically studied applications of any herbal medicine, with standardised extracts shown comparable to conventional antidepressants for mild-to-moderate depression.[1, 4]

Chickweed has a long European and North American folk-medicine tradition as a cooling, soothing emollient for itchy, inflamed skin conditions such as eczema, and as a nutritious wild spring green. Modern phytochemical review confirms broad anti-inflammatory and antioxidant activity, though clinical trial evidence remains limited.[1, 11]

Preparations

Standardised extract[1]

Flowering-top extract standardised to hypericin and/or hyperforin content, taken as tablets or capsules; the best-studied clinical form for mood support.

Fresh leaf poultice/salve[11]

Crushed fresh herb, or an infused oil/salve, applied to itchy, irritated or inflamed skin.

Dosage

Standardised extract[1]

Clinical trials for mild-to-moderate depression commonly use around 300 mg of standardised extract three times daily (900 mg/day total); given extensive drug interactions, use should be discussed with a healthcare provider, especially alongside other medicines. Educational reference only, not a prescription.

Topical salve / infusion[11, 12, 13]

Chickweed is generally used topically as a salve or poultice for skin irritation, or as a mild infusion (roughly 2-4 g dried herb per cup); large quantities as food should be moderated given its oxalate content. Educational reference only, not a prescription.

Drug Class Interactions

Safety note[18, 19, 20, 21]Avoid
Drug Class: antidepressants-serotonergic
Mechanism: St John's wort raises serotonin activity; combined with SSRIs, SNRIs or MAOIs it can trigger serotonin syndrome (agitation, tremor, sweating, rapid heartbeat). Reviews of clinical reports document serotonin syndrome and lethargy when it is combined with serotonin-reuptake inhibitors.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03
Safety note[18, 19, 22]Avoid
Drug Class: antiretrovirals
Mechanism: Potent CYP3A4 and P-glycoprotein induction lowers antiretroviral levels (indinavir exposure fell ~57%), risking loss of viral control and drug resistance.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03
Safety note[18, 19, 23]Avoid
Drug Class: immunosuppressants
Mechanism: Enzyme and transporter induction reduces ciclosporin and tacrolimus levels; reported to cause subtherapeutic concentrations and transplant rejection.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03
Safety note[18, 24, 25, 26]Avoid
Drug Class: hormonal-therapies
Mechanism: Increased metabolism of ethinylestradiol and progestins reduces contraceptive exposure, causing breakthrough bleeding, ovulation and unplanned pregnancy. Randomised and controlled trials in women confirmed more breakthrough bleeding, reduced progestin levels and evidence of ovulation when St John's wort was added to the pill.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03
Safety note[18, 19, 21]Caution
Drug Class: anticoagulants-antiplatelets
Mechanism: CYP induction increases warfarin clearance and can lower INR, reducing the anticoagulant effect; close monitoring is needed.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03
Safety note[18, 27]Caution
Drug Class: cardiac-glycosides
Mechanism: P-glycoprotein induction lowers digoxin levels (AUC fell ~25% over ten days), which may reduce its effect.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03
Safety note[18, 19, 21]Caution
Drug Class: statins
Mechanism: CYP3A4 induction lowers levels of simvastatin and atorvastatin, potentially weakening their cholesterol-lowering effect.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03
Safety note[18, 19, 21]Caution
Drug Class: cyp3a4-substrates
Mechanism: As a broad CYP3A4 and P-glycoprotein inducer, St John's wort can lower levels of many medicines cleared by this pathway; check each medication individually.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03
Safety note[20, 28, 29]Avoid
Drug Class: chemotherapy-agents
Mechanism: St John's wort strongly induces CYP3A4 and P-glycoprotein, speeding the breakdown and removal of several cancer medicines. In patients it cut the active form of irinotecan (SN-38) by about 42% and reduced imatinib exposure by roughly a third - enough to weaken treatment and risk drug resistance. Do not take St John's wort during chemotherapy.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03

Not documented

References

REF-0842, REF-0843, REF-0844, REF-1789, REF-1790, REF-1791, REF-1792, REF-1793, REF-1794, REF-1795, REF-1796, REF-1797, REF-1798, REF-1799
REF-0900, REF-0901, REF-0902, REF-0903, REF-0904, REF-0905, REF-0906, REF-0907, REF-0908, REF-0909

Pairings

St John's wort raises serotonin activity and can cause serotonin syndrome when combined with other serotonin-boosting agents; pairing it with another mood-active, serotonergic herb such as saffron may add to this risk.[30]

Partner Id: crocus-sativus
Type: caution
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03

St John's wort is a strong enzyme inducer and increases serotonin activity; combining it with another antidepressant-type herb such as rhodiola may add to serotonergic effects and unpredictably change how each is handled by the body.[30]

Partner Id: rhodiola-rosea
Type: caution
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03

St John's wort speeds up the breakdown of many substances and also raises serotonin activity; pairing it with a sedative herb such as valerian may add to drowsiness and can unpredictably change how each is handled by the body.[30, 31]

Partner Id: valeriana-officinalis
Type: caution
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03

St John's wort is a strong enzyme inducer and kava is a liver-metabolised sedative; combining them may add to central-nervous-system effects and alter how kava is processed, so combined use warrants caution.[30, 31]

Partner Id: piper-methysticum
Type: caution
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-03

Not documented

Lookalikes Review

Outcome: none-known
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-07
Outcome: has-lookalikes
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-06

Dangerous Lookalikes

Not documented

Safety note[15, 16, 17, 18]Irritant
Dangerous Plant: euphorbia-peplus
Confused Part: Whole above-ground plant (stems and leaves) gathered raw from the same low, weedy, disturbed ground where chickweed is picked. Every part carries the caustic milky latex.
Confusion Context: Petty spurge (radium weed, Euphorbia peplus) is a small sprawling annual of gardens, paths and disturbed soil - the exact habitat chickweed is foraged from - and is repeatedly named by foraging authorities as the poisonous lookalike of chickweed. The hazard is chemical injury from its latex: the diterpene-ester sap (ingenol mebutate, confirmed in E. peplus by Ramsay et al. 2011) is caustic to skin, lips, mouth and especially the eye. Euphorbia latex in the eye behaves like an alkaline chemical burn and has caused keratouveitis, corneal epithelial defects, hypopyon and temporary vision loss in published case reports (Merani et al. 2007; Fleischman et al. 2012). Breaking a plant open in the hand and eating it transfers this caustic sap to lips, mouth and gut.
Distinguishing Features: SNAP TEST (decisive): petty spurge instantly bleeds a white MILKY latex from any broken stem or leaf. Chickweed NEVER produces milky sap - torn chickweed shows only clear juice and a stretchy elastic inner thread., Stem: petty spurge has a smooth, round, hairless stem. Chickweed has one distinct line of fine white hairs running up just one side of the stem, switching sides at each leaf node., Flowers: petty spurge has tiny green cup-shaped flower structures (cyathia) with no true petals. Chickweed has small white star flowers whose five petals are so deeply cleft they look like ten.
Key Test: Break a stem. If a white milky latex oozes out it is spurge - discard it and keep it away from mouth and eyes. Chickweed yields only clear juice and a stretchy inner thread, never milk.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-06
Safety note[18, 19, 20]Irritant
Dangerous Plant: lysimachia-arvensis
Confused Part: Whole leafy plant (stems and leaves) picked raw from cultivated or weedy ground, especially before flowering; all parts contain the toxic saponins.
Confusion Context: Scarlet pimpernel (Lysimachia arvensis, syn. Anagallis arvensis) is a low sprawling annual of the same disturbed weedy ground where chickweed is gathered and shares the small-leaved trailing habit, so pre-flowering shoots can be mistaken for chickweed. It contains haemolytic saponins; the University of California notes it can be toxic to livestock and humans, with toxicity ranging from virtually nontoxic to fatally toxic. Genuine lethal potential is on record: Schneider (1978) experimentally reproduced fatal nephrosis with uraemia and renal cortical necrosis by dosing sheep with the plant. The realistic human outcome from eating it in a salad is gastrointestinal upset, but it is not food.
Distinguishing Features: Stem: scarlet pimpernel has a hairless stem that is distinctly SQUARE (four-angled) in cross-section, with no line of hairs. Chickweed has a rounded stem bearing one line of fine hairs up a single side that switches sides at each node., Leaves: pimpernel leaves are stalkless (sessile) and clasp the square stem in opposite pairs. Chickweed's lower leaves have a clear stalk (petiole)., Flowers: pimpernel flowers are salmon-orange (occasionally brick-red, blue or white) with five rounded un-notched petals. Chickweed flowers are white with five deeply cleft petals looking like ten. Any salmon-orange flower rules out chickweed., Neither plant has milky sap, so the milky-latex test does NOT separate chickweed from pimpernel - use the square hairless stem, sessile opposite leaves and orange flowers instead.
Key Test: Roll the stem between your fingers. Pimpernel's stem is square-edged and hairless all round; chickweed's rounded stem carries a single line of hairs on one side that jumps sides at each node. If flowers are present, any salmon-orange flower means it is not chickweed.
Reviewed By: Omnia Sana (owner-authorized)
Reviewed Date: 2026-07-06

References & Sources

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  2. Kholghi, G., Arjmandi-Rad, S., Zarrindast, M.R. and Vaseghi, S (2022) 'St. John's wort (Hypericum perforatum) and depression: what happens to the neurotransmitter systems?', Naunyn-Schmiedeberg's Archives of Pharmacology, 395(6), pp. 629-642. doi:10.1007/s00210-022-02229-z Traditional / reference
    https://doi.org/10.1007/s00210-022-02229-z
  3. Fugh-Berman, A (2000) 'Herb-drug interactions', Lancet, 355(9198), pp. 134-138. doi:10.1016/S0140-6736(99)06457-0 Traditional / reference
    https://doi.org/10.1016/S0140-6736(99)06457-0
  4. Nobakht, S.Z., Akaberi, M., Mohammadpour, A.H., Tafazoli Moghadam, A. and Emami, S.A (2022) 'Hypericum perforatum: Traditional uses, clinical trials, and drug interactions', Iranian Journal of Basic Medical Sciences, 25(9), pp. 1045-1058. doi:10.22038/IJBMS.2022.65112.14338 Meta-analysis / review
    https://doi.org/10.22038/IJBMS.2022.65112.14338
  5. Jiang, Z., Wang, F., Zhao, Y., Lu, L., Jiang, X., Huang, T., Lin, Y., Guo, L., Weng, Z. and Liu, E (2024) 'Hypericum perforatum L. attenuates depression by regulating Akkermansia muciniphila, tryptophan metabolism and NFkB-NLRP2-Caspase1-IL1beta pathway', Phytomedicine, 132, pp. 155847. doi:10.1016/j.phymed.2024.155847 Preclinical
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  7. Russo, E., Scicchitano, F., Whalley, B.J., Mazzitello, C., Ciriaco, M., Esposito, S., Patane, M., Upton, R., Pugliese, M., Chimirri, S., Mammi, M., Palleria, C. and De Sarro, G (2013) 'Hypericum perforatum: pharmacokinetic, mechanism of action, tolerability, and clinical drug-drug interactions', Phytotherapy Research, 28(5), pp. 643-655. doi:10.1002/ptr.5050 Meta-analysis / review
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  10. Liu, Y., Jiang, Y., Huang, R., Yang, J., Xiao, B. and Dong, J (2013) 'Hypericum perforatum L. preparations for menopause: a meta-analysis of efficacy and safety', Climacteric, 17(4), pp. 325-335. doi:10.3109/13697137.2013.861814 Meta-analysis / review
    https://doi.org/10.3109/13697137.2013.861814
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  13. Nathan, P (1999) 'The experimental and clinical pharmacology of St John's Wort (Hypericum perforatum L.)', Molecular Psychiatry, 4(4), pp. 333-338. doi:10.1038/sj.mp.4000557 Meta-analysis / review
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  14. Verotta, L (2003) 'Hypericum perforatum, a source of neuroactive lead structures', Current Topics in Medicinal Chemistry, 3(2), pp. 187-201. doi:10.2174/1568026033392589 Meta-analysis / review
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  15. Linde, K. et al (2008) 'St John'. Traditional / reference
    https://scholar.google.com/scholar?q=St%20John
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    https://scholar.google.com/scholar?q=Handbook%20of%20Medicinal%20Herbs%2C%20Second%20Edition
  18. Zhou, S., Chan, E., Pan, S.Q., Huang, M. and Lee, E.J.D (2004) 'Pharmacokinetic interactions of drugs with St John's wort', Journal of Psychopharmacology, 18(2), pp. 262-276. doi:10.1177/0269881104042632 Meta-analysis / review
    https://doi.org/10.1177/0269881104042632
  19. Izzo, A.A. and Ernst, E (2009) 'Interactions between herbal medicines and prescribed drugs: an updated systematic review', Drugs, 69(13), pp. 1777-1798. doi:10.2165/11317010-000000000-00000 Meta-analysis / review
    https://doi.org/10.2165/11317010-000000000-00000
  20. Borrelli, F. and Izzo, A.A (2009) 'Herb-drug interactions with St John's wort (Hypericum perforatum): an update on clinical observations', The AAPS Journal, 11(4), pp. 710-727. doi:10.1208/s12248-009-9146-8 Meta-analysis / review
    https://doi.org/10.1208/s12248-009-9146-8
  21. Nicolussi, S., Drewe, J., Butterweck, V. and Meyer zu Schwabedissen, H.E (2020) 'Clinical relevance of St. John's wort drug interactions revisited', British Journal of Pharmacology, 177(6), pp. 1212-1226. doi:10.1111/bph.14936 Meta-analysis / review
    https://doi.org/10.1111/bph.14936
  22. Piscitelli, S.C., Burstein, A.H., Chaitt, D., Alfaro, R.M. and Falloon, J (2000) 'Indinavir concentrations and St John's wort', Lancet, 355(9203), pp. 547-548. doi:10.1016/S0140-6736(99)05712-8 Clinical study
    https://doi.org/10.1016/S0140-6736(99)05712-8
  23. Barone, G.W., Gurley, B.J., Ketel, B.L., Lightfoot, M.L. and Abul-Ezz, S.R (2000) 'Drug interaction between St. John's wort and cyclosporine', Annals of Pharmacotherapy, 34(9), pp. 1013-1016. doi:10.1345/aph.10088 Clinical study
    https://doi.org/10.1345/aph.10088
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Generated automatically from the Omnia Sana plant database and its cited sources. For educational purposes only — not medical advice. Always consult a qualified practitioner before using medicinal plants.